Abstract P093: Psychological Stress is Associated with Increases in Weight and Waist Circumference over Time: Longitudinal Data from the Dallas Heart Study
Background: Psychological stress (stress) is associated with cardiovascular events, but the impact of stress on changes in adiposity as a measure of cardio-metabolic risk is unknown.
Methods and Results: We examined the relationship between self-reported psychological stress level at baseline and change in weight and waist circumference (WC) over a median 7-year interval in the Dallas Heart Study, a multiethnic, population-based sample of Dallas County residents aged 18-65 (N = 1603; whites = 615; blacks = 754; Hispanics = 234). Participants answered a questionnaire and underwent measurement of weight, height and WC at baseline and follow-up visits between 2000 and 2009. Stress levels were determined by self-report using a five-point scale, with responses categorized as low (1-2), moderate (3) and high (4-5) stress levels. Linear regression modeling was used to determine the association between baseline stress category and weight/WC change over time. At baseline, 24% of participants reported low stress, 49% moderate, and 27% high stress. No significant differences were observed in weight, BMI or WC at baseline between stress groups. Compared to participants reporting high stress, those with low stress were older (45 vs. 43 yrs), more likely to be male (54% vs. 37%) and be of black race (55% vs. 34%) [p<0.01 for all]. In unadjusted modeling, those in the high stress group gained more weight and had greater WC increase compared to the low stress group (Table). After adjusting for age, sex, baseline adiposity, race, education and income, the high stress group still had greater weight gain compared to the low stress group.
Conclusions: Higher levels of self-reported stress are associated with adverse changes in adiposity among DHS participants. These results suggest that interventions targeting psychological stress may lessen the impact of obesity on subsequent cardio-metabolic risk.
Author Disclosures: R. Cooper-McCann: B. Research Grant; Significant; National Institutes of Health (NIH) Medical Research Scholars Program - public-private partnership supported jointly by NIH and generous contributions to the Foundation for the NIH (FNIH), contributions to the FNIH from Pfizer Inc., contributions to the FNIH from The Doris Duke Charitable Foundation, contributions to the FNIH from The Alexandria Real Estate Equities, Inc, contributions to the FNIH from Mr. and Mrs. Joel S. Marcus, contributions to the FNIH from the Howard Hughes Medical Institute. C. Ayers: None. M. Albert: None. J. de Lemos: None. I. Neeland: None. M. McClurkin: None. E. Shropshire: None. T.M. Powell-Wiley: None.
- © 2014 by American Heart Association, Inc.