From the Department of Cardiology/MMCC, Wilford Hall Medical Center,
Lackland AFB, Tex (W.J.L.) and the Department of Cardiology/MCHE-MDC, Brooke
Army Medical Center, Fort Sam Houston, Tex (T.A.C.).
Correspondence to William J. Lauer, MD, Major, USAF, MC, Department of Cardiology, 375th Medical Group, 310 W. Losey St, Scott AFB, IL 62225-5252.
An 84-year-old woman
with exertional chest pain underwent a preoperative evaluation for a
colectomy. Echocardiography revealed hyperdynamic
left ventricular systolic function and moderate
concentric left ventricular hypertrophy.
Adenosine 201Tl single photon emission CT
(SPECT) imaging demonstrated a reversible anterior wall perfusion
defect. At coronary arteriography, her coronary
arteries appeared to be free of atherosclerotic disease; however,
systolic bridging of the mid left anterior descending
coronary artery (LAD) was observed.
Figures 1
The patient was started on a ß-blocker with subsequent resolution of
her chest pain and underwent a successful colectomy without suffering a
cardiac event.
Footnotes
The editor of Images in Cardiovascular Medicine is Hugh A. McAllister, Jr, MD, Chief, Department of Pathology, St Luke's Episcopal Hospital and Texas Heart Institute, and Clinical Professor of Pathology, University of Texas Medical School and Baylor College of Medicine.
Circulation encourages readers to submit cardiovascular images to Dr Hugh A. McAllister, Jr, St Luke's Episcopal Hospital and Texas Heart Institute, 6720 Bertner Ave, MC1267, Houston, TX 77030.
© 1998 American Heart Association, Inc.
Images in Cardiovascular Medicine
Myocardial Bridging
and 2
from her coronary arteriogram
demonstrate myocardial bridging. In Figure 1
, systolic
compression of the midportion of the LAD is seen, with return to a
normal caliber during diastole seen in Figure 2
. Figure 3
shows the 201Tl
SPECT images. On these verticle long-axis images, a partial perfusion
defect of the anterior wall is noted with adenosine stress that
reverses almost completely with rest.

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Figure 1.

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Figure 2.

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Figure 3.
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