Circulation. 1997;95:2327-2328
(Circulation. 1997;95:2327-2328.)
© 1997 American Heart Association, Inc.
Reversible Tachycardia-Induced Cardiomyopathy
Anwer Dhala, MD;
John P. Thomas, MD
From Children's Hospital of Wisconsin, Milwaukee.
Correspondence to Anwer Dhala, MD, 2901 W K.K. River Pkwy, No. 470, Milwaukee, WI 53215-3660.
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Introduction
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The boy was treated with oxygen, diuretics, digoxin,
afterload
reduction, and anticoagulation. Despite improvement in
symptoms,
he remained persistently tachycardic, with variable AV
block
during sleep. Amiodarone therapy was initiated for
presumed
automatic atrial tachycardia. Because
tachycardia remained uncontrolled,
he underwent an
electrophysiological study, and a single
right
atrial tachycardia focus was ablated (Fig 3

); he has remained
in sinus rhythm since then. Gradual
but complete resolution
of his cardiac dysfunction was noted over a 12-
to 16-week period.

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Figure 3. Tracing after ablation.
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Figure 1. Electrocardiogram.
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Figure 2. Transthoracic echocardiogram.
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Footnotes
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The editor of Images in Cardiovascular Medicine is Hugh A. McAllister,
Jr, MD, Chief, Department of Pathology, St Luke's Episcopal
Hospital and Texas Heart Institute, and Clinical Professor of
Pathology, University of Texas Medical School and Baylor College
of Medicine.
Circulation encourages readers to submit cardiovascular images to Dr Hugh A. McAllister, Jr, St Luke's Episcopal Hospital and Texas Heart Institute, 6720 Bertner, MC 4-265, Houston, TX 77030.
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U. C Hoppe and D. J Beuckelmann
Characterization of the hyperpolarization-activated inward current in isolated human atrial myocytes
Cardiovasc Res,
June 1, 1998;
38(3):
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[Abstract]
[Full Text]
[PDF]
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