(Circulation. 2007;115:2750-2760.)
© 2007 American Heart Association, Inc.
Interventional Cardiac Electrophysiology |
From the Department of Internal Medicine, Cardiovascular Division, Brigham and Womens Hospital, Boston, Mass (W.G.S.), and Cardiovascular Division, Department of Internal Medicine, Keio University Hospital, Tokyo, Japan (K.S.).
Correspondence to William G. Stevenson, MD, Cardiovascular Division, Brigham and Womens Hospital, 75 Francis St, Boston, MA 02115. E-mail wstevenson@partners.org
Key Words: ablation arrhythmia cardiomyopathy myocardial infarction tachyarrhythmias
An extract of the first 250 words of the full text is provided, because this article has no abstract. |
| Introduction |
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20% of patients within 3 to 5 years after ICD implantation for primary prevention of sudden death in high-risk groups.24 ICD shocks reduce quality of life and are associated with an increased risk of death.24 Antiarrhythmic drug therapy with amiodarone or sotalol reduces VT episodes but with disappointing incidence of side effects and efficacy.2 Catheter ablation is useful for reducing VT episodes and can be life-saving when VT is incessant.1,5,6 Idiopathic VTs occur in patients without structural heart disease and rarely cause sudden death. Electrophysiological study with catheter ablation is often warranted to confirm the diagnosis, to provide further evidence for the absence of ventricular scar or other disease, and often to cure the arrhythmia. Ablation is also an option for symptomatic nonsustained VT and frequent ventricular ectopy in these patients.1
The appearance of the VT on ECG often suggests its likely cause and associated heart disease (Figure 1). Monomorphic VT has the same QRS complex from beat to beat, indicating repetitive ventricular activation from a structural substrate or focus that can be targeted for ablation. Most are due to reentry through regions of ventricular scar.7
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