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(Circulation. 2005;112:1663-1675.)
© 2005 American Heart Association, Inc.

New Drugs and Technologies

Endovascular Repair of Abdominal and Thoracic Aortic Aneurysms

Barry T. Katzen, MD; Michael D. Dake, MD; Alexandra A. MacLean, MD; David S. Wang, MD

From the Baptist Cardiac and Vascular Institute (B.T.K., A.A.M.), Miami, Fla; University of Virginia Health System (M.D.D.), Charlotteville; and Stanford University School of Medicine (D.S.W.), Stanford, Calif.

Correspondence to Barry T. Katzen, MD, Founder and Medical Director, 8900 N Kendall Dr, Miami, FL 33176-2197. E-mail barryk@baptisthealth.net

An extract of the first 250 words of the full text is provided, because this article has no abstract.

	Introduction

 
Aortic aneurysms remain a challenging problem for patients and physicians. There have been major advances in the treatment of large-vessel aneurysms during the past 10 years. The surgical armamentarium used to treat these aneurysms now includes an endovascular approach that allows the insertion of a graft to exclude the aneurysm sac from blood flow. The endovascular repair of abdominal and thoracic aortic aneurysms has become a viable alternative to open repair and is often the approach of choice for high-risk patients. In this review, we examine the endovascular treatment of abdominal and thoracic aortic aneurysms.

	Endovascular Repair of Abdominal Aortic Aneurysms

 
Abdominal aortic aneurysms (AAAs) are a formidable diagnosis for patients. This is a life-threatening condition that mandates consideration of repair. A ruptured AAA has a mortality rate approaching 90%; however, when an AAA is repaired electively, the mortality drops to less than 5%.^1–3 There is, therefore, a clear advantage to treating these aneurysms before they rupture. Because this disease affects 4% to 7% of adults over the age of 65 years, with a far greater prevalence in males than females, clinicians will encounter this problem more frequently as the population ages.⁴

AAAs usually develop in patients with a history of arteriosclerosis or smoking. Patients present for repair when it is discovered that there is a dilation of their abdominal aorta to a diameter 1.5 times normal. The result is a weakened aortic wall that is at increased risk of rupture. The pathogenesis of this aortic wall change likely involves enzymes responsible for elastin and collagen . . . [Full Text of this Article]

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