(Circulation. 2004;109:e222-e223.)
© 2004 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Department of Internal Medicine I and Cardiology (G.O.K., C.H., T.B.) and Department of Radiology and Neuroradiology (N.R., G.A., D.K.), Alfried Krupp Krankenhaus, and Department of Internal Medicine, Cardiology Clinic, Kliniken Essen-Sued (A.L., B.K.), Essen, Germany.
Correspondence to Gert O. Kerkhoff, MD, EC, Department of Internal Medicine I and Cardiology, Alfried Krupp Krankenhaus, Essen, Alfried Krupp Strasse 21, 45117 Essen, Germany.
An extract of the first 250 words of the full text is provided, because this article has no abstract. |
A 66-year-old woman (169 cm, 70 kg) with known 3-vessel coronary artery disease, a mildly reduced left ventricular (LV) ejection fraction (55% by echocardiography), and a history of anterior myocardial infarction (1997) underwent routine echocardiography that showed a thrombus-like, mobile formation at the apex. Transthoracic and transesophageal echocardiography did not clarify the situation. Consequently, contrast-enhanced cardiac MRI was performed to evaluate LV function, depict size of the myocardial infarction, and differentiate apical pathology using a 1.5-T scanner (Siemens Magnetom Sonata Maesto Class).
Horizontal long-axis breath-hold cine-MR (TRUE-Fisp) showed akinetic apical and septal segments with apical wall thinning and no typical myocardium. Between the LV chamber and the apex, a membranous border with signs of turbulence ("voids") indicates a communication between the left ventricle and a "chronic" pseudoaneurysm (Figures 1 and 2
). T1-weighted (Segmented Inversion recovery TurboFLASH) image acquisition showed transmural scarring in the anteroapical segments (Figure 3). There were no signs of thrombus formation.
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The editor of Images in Cardiovascular Medicine is Hugh A. McAllister, Jr, MD, Chief, Department of Pathology, St Lukes Episcopal Hospital and Texas
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