Donate Help Contact The AHA Sign In Home
American Heart Association
Circulation
Search: search_blue_button Advanced Search
Circulation. 2003;107:2761-2763
doi: 10.1161/01.CIR.0000067884.98471.0A
This Article
Right arrow Full Text
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowRequest Permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Reddy, V. Y.
Right arrow Articles by Ruskin, J. N.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Reddy, V. Y.
Right arrow Articles by Ruskin, J. N.
Related Collections
Right arrow Congestive
Right arrow Pacemaker

(Circulation. 2003;107:2761.)
© 2003 American Heart Association, Inc.


Images in Cardiovascular Medicine

Electroanatomic Mapping of Cardiac Resynchronization Therapy

Vivek Y. Reddy, MD; Petr Neuzil, MD; Milos Taborsky, MD; Stepan Kralovec;; Lucie Sedivá, MD; Jeremy N. Ruskin, MD

From the Cardiac Arrhythmia Services of Massachusetts General Hospital–Harvard Medical School, Boston, Mass (J.N.R., V.Y.R.), and Na Homolce Hospital, Prague, Czech Republic (P.N., M.T., S.K., L.S.).

Correspondence to Vivek Y. Reddy, MD, Cardiac Arrhythmia Service, Massachusetts General Hospital, 55 Fruit St, GRB 109, Boston, MA 02114. E-mail vreddy@partners.org


An extract of the first 250 words of the full text is provided, because this article has no abstract.
 

A 51-year-old man with a history of dilated cardiomyopathy presented with New York Heart Association Class III congestive heart failure. Despite optimal medical management, he continued to experience symptoms consistent with low-output failure: exertional dyspnea, orthopnea, and paroxysmal nocturnal dyspnea. He was hospitalized 3 times in the previous 4 months for congestive heart failure management. The baseline ECG revealed a left bundle-branch block with a QRS width of 172 ms, and the echocardiogram revealed an ejection fraction of 28%. He was referred to the cardiac arrhythmia service for cardiac resynchronization therapy. A biventricular pacemaker was implanted in the left prepectoral region. A transvenous approach was utilized to position both the standard right-sided leads, and the left ventricular lead was positioned via the coronary sinus (Figure 1).


Figure Removed (Available Only in the Full Text)
View larger version (137K):
[in this window]
[in a new window]
 
Figure 1. Fluoroscopic position of the cardiac pacing leads. The pacing leads were positioned at the right atrial appendage (RAA), right ventricular apex (RV) and a posterolateral branch of the coronary sinus (LV). The unipolar LV pacing lead was placed by using a transvenous approach after retrogradely engaging the ostium of the coronary sinus with a long sheath.

Two months after implantation of the biventricular pacing system, the patient underwent electroanatomic mapping of both ventricles during an electrophysiological study. Electroanatomic mapping revealed a significant change in the electrical activation pattern during either right ventricular (Figure 2) or biventricular (Figure 3) pacing. As compared with right ventricular pacing alone (Figure 2), biventricular pacing resulted in a concomitant . . . [Full Text of this Article]