(Circulation. 2002;106:176.)
© 2002 American Heart Association, Inc.
Clinician Update |
From the Cardiovascular Division, Department of Internal Medicine, Brigham and Womens Hospital, and Harvard Medical School, Boston, Mass.
Correspondence to William G. Stevenson, MD, Cardiovascular Division, Brigham and Womens Hospital 75 Francis St, Boston, MA 02115. E-mail wstevenson@partners.org
An extract of the first 250 words of the full text is provided, because this article has no abstract. |
| Introduction |
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She was referred for catheter ablation. An echocardiogram revealed akinesis of the inferior wall and no left ventricular thrombus. In the electrophysiology laboratory, programmed stimulation induced 5 different morphologies of VT (Figure 1) with rates ranging from 180 to 220 bpm. Because the induced VTs were unstable, producing hypotension and often changing from one VT to another, catheter mapping and ablation were performed largely during sinus rhythm, guided by electrogram characteristics and pacing during sinus rhythm (pace-mapping) that marked the location of the infarct scar and likely reentry paths in the subendocardium. After placement of lines of radiofrequency (RF) lesions through these abnormal regions, only ventricular flutter (280 bpm) was inducible; the slower VTs were no longer inducible. There have been no VT recurrences in the 18 months
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