(Circulation. 2001;103:161.)
© 2001 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Cardiovascular Center, Echocardiography Laboratory, University Hospital, Zurich, Switzerland.
Correspondence to Andre Linka, MD, Cardiovascular Center, Echocardiography Laboratory, University Hospital, Raemistrasse 100, 8091 Zurich, Switzerland. E-mail karlinka@usz.unizh.ch
A 65-year-old
woman with terminal liver disease and portal hypertension secondary to
alcohol-induced cirrhosis underwent an elective transjugular
intrahepatic portosystemic shunt procedure. A self-expanding,
100x80-mm Nitinol (Jomed AG) stent was implanted and extended
cranially using an additional 40x12-mm Wallstent (Boston
Scientific). During the same admission, she was evaluated and
accepted for liver transplantation, which was performed 4 days later
when an organ was available. In the explanted liver, one patent
intraparenchymal stent was described by the pathologist. Three weeks
after surgery, she was referred to a rehabilitation facility. The new
organ showed good function, but she had a recurrence of ascites that
was managed medically, and she returned home 6 weeks after liver
transplantation. In the seventh postoperative week, however, the amount
of ascites increased, which necessitated the drainage of several
liters of peritoneal fluid. About 8 weeks after
surgery,she became increasingly dyspneic. A chest
radiograph at that time showed no evidence of pleural effusion or
consolidation but, surprisingly, it did reveal a radio-opaque
stent-like structure projected over the right ventricle. Doppler
echocardiography
(Figure 1
and 2
) was performed, and it revealed a stent in
the right ventricle caught within the subvalvular tricuspid apparatus,
leading to severe tricuspid regurgitation.
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