(Circulation. 2000;102:937.)
© 2000 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Arrhythmia Service of the Albert Einstein College of Medicine, Montefiore Medical Center, New York, NY.
Correspondence to Eugen C. Palma, MD, Albert Einstein College of Medicine, Montefiore Medical Center, Arrhythmia Service, 111 East 210th St, Bronx, NY 10467. E-mail eupalma@montefiore.org
A37-year-old man with frequent episodes of
paroxysmal atrial fibrillation refractory to antiarrhythmic therapy
(and no other documented arrhythmias on multiple Holter
examinations) underwent an attempt at radiofrequency ablation of the
atrial fibrillation. Catheter positions are shown in the left anterior
oblique view in Figure 1
and include 2
catheters in the left and right upper pulmonary veins. During
mapping, the patient developed atrioventricular node
reentry tachycardia (AVNRT) both spontaneously and with
atrial extrastimuli; this AVNRT was later ablated. Before ablation, the
patients rhythm spontaneously changed from AVNRT to atrial
fibrillation (Figure 2
). The catheter in
the right upper pulmonary vein clearly recorded
pulmonary venous potentials, which initiated atrial ectopic
beats (as seen in the change of the activation sequence in the
coronary sinus) and subsequent atrial fibrillation.
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