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(Circulation. 2000;102:713.)
© 2000 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Department of Medicine, University Hospital, CH-8091 Zürich, Switzerland.
Correspondence to Christoph Scharf, MD, Division of Cardiology, Department of Medicine, University Hospital, CH-8091 Zürich, Switzerland. E-mail christoph.scharf@dim.usz.ch
A36-year-old man who had undergone splenectomy several
years earlier as a result of trauma was admitted with malaise and
scattered hemorrhagic lesions on his nose
(Figure
, A). Two days earlier, his dog
had bitten him. The next day, he developed chills, watery diarrhea, and
vomiting. On admission, the patient complained of chest discomfort. On
physical examination, his core temperature was 37.8°C, his blood
pressure was 80/60 mm Hg, his heart rate was 90 beats/min, and
his respiration was 10 breaths/min, with normal cardiopulmonary
auscultation. At the site of the dog bite, a discrete lesion was
visible, without any sign of infection (Figure
, B). The surface
ECG showed ST elevation in the inferolateral leads (Figure
, C).
Laboratory examination revealed a white blood cell count of 32 400
cells/µL, a C-reactive protein level of 549 mg/dL, a
creatinine level of 268 µmol/L, disseminated
intravascular coagulation with thrombocytopenia of 31 000 cells/µL,
and D-dimers of 10.2 mg/L. Creatine kinase levels rose from 788 IU/L to
3029 IU/L after 6 hours, and they peaked at 3306 IU/L after 12
hours. The troponin-I concentration rose from 1.2 µg/L to 108 µg/L
after 6 hours and declined to 38 µg/L on the second hospital day.
|
Diagnostic procedures included a thorough examination of
the peripheral
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