(Circulation. 1999;100:564-565.)
© 1999 American Heart Association, Inc.
Images in Cardiovascular Medicine |
From the Minneapolis Heart Institute at Abbott Northwestern Hospital (J.H.T., J.R.L., B.P.F., D.M.N., T.F., R.G.H.), the Department of Internal Medicine (O.M.O.), the Department of Pathology (C.A.H.), and the Cardiovascular Division (J.H.T.), University of Minnesota Medical School, Minneapolis, and Mille Lacs Health System, Onamia, Minn (T.H.B.).
Correspondence to Jay H. Traverse, MD, Minneapolis Heart Institute at Abbott Northwestern Hospital, Minneapolis Cardiology Associates, 920 E 28th St, Suite 300, Minneapolis, MN 55407. E-mail trave004@tc.umn.edu
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The ECG showed normal sinus rhythm, left atrial enlargement, incomplete right bundle-branch block, and voltage and T-wave changes suggestive of left ventricular hypertrophy. An echocardiogram showed a global decrease in left ventricular function, with an ejection fraction of 25%. There was mild left ventricular hypertrophy and mild mitral regurgitation. The chest radiograph showed moderate cardiomegaly with clear lung fields and normal pulmonary vasculature. Significant laboratory findings included a hemoglobin of 10.1 (mean corpuscular volume=90) and an elevated calcium of 11.6. The patient was referred for bilateral heart catheterization and coronary angiography.
At angiography, her femoral pulses were diminished. A guidewire could
not be passed beyond the thoracic aorta; therefore, angiography was
performed with a left brachial approach. The proximal aortic
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