(Circulation. 1995;91:2284-2289.)
© 1995 American Heart Association, Inc.
Articles |
From the Texas Heart Institute (W.E.D.), St Luke's Episcopal Hospital, and the Departments of Radiology (P.C.), Internal Medicine (Division of Cardiology) (E.B., H.V.A., A.T.V.), and Surgery (M.P.M.), The University of Texas Medical School at Houston, The University of TexasHouston Health Science Center, Houston, Tex.
Correspondence to Herbert L. Fred, MD, Clinical Editor of Clinicopathological Conferences, Circulation Editorial Offices, St Luke's Episcopal Hospital/Texas Heart Institute, 6720 Bertner Ave, Room B524 (MC1-267), Houston, TX 77030-2697.
| Case Presentation |
|---|
The patient initially improved in the emergency department while lying supine with an O2 saturation of 97% on 2 L/min O2 by nasal cannula. However, when she stood up to make a telephone call, she noted acute increase in dyspnea, and her O2 saturation fell to the low 80s and was minimally corrected with increased oxygen supplementation. Consequently, she was admitted to the intensive care unit for further evaluation and treatment.
She had had one previous episode of prolonged shortness of breath and
fatigue approximately 3 months earlier while raking leaves in the hot
summer sun. She attributed her symptoms to the heat. Otherwise, she had
been fairly active at home, was able to perform daily chores without
dyspnea or chest pain, denied paroxysmal nocturnal dyspnea or
orthopnea, and noted only mild swelling of her ankles occasionally. She
had no dyspnea or cyanotic spells during childhood and had tolerated
two pregnancies without difficulty. She denied hemoptysis or pleuritic
chest pain, had
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