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(Circulation. 2003;107:e9007.)
© 2003 American Heart Association, Inc.

Cardiovascular News

Ruth SoRelle, MPH

Circulation Newswriter

An extract of the first 250 words of the full text is provided, because this article has no abstract.

Manuscript Submissions to Circulation Set Record

In December 2002, the editorial offices of Circulation received 670 manuscripts, the most ever in a single month.

"This is an indication of the importance of research into cardiovascular disease around the world and the need for a weekly journal such as Circulation to insure the prompt dissemination of that information," said James T. Willerson, MD, Editor of Circulation. "Circulation has continued to receive reports of some of the best research ongoing in cardiovascular medicine today, and its ability to get that information to the clinician and the research community in a timely fashion has prompted many of those in the field to use it as a medium for communicating their results. As the journal continues, we expect that it will continue to reflect advances in the continuing war on cardiovascular disease worldwide."

Nicotine and Its Ways
Researchers from Germany and California answered "yes" to the logical question: "If antigen-presenting cells play a role in adaptive immunity and this plays a role in the growth of atherosclerotic plaque, and nicotine increases the growth of atherosclerotic lesions, does nicotine stimulate antigen-producing cells and their ability to stimulate T-cells?"

In this week’s issue of Circulation (Circulation. 2003;107:604–611), scientists led by Alexandra Aicher, MD, and Christopher Heeschen, MD, of the Department of Internal Medicine IV, Molecular Cardiology at the University of Frankfurt, Germany, used dendritic cells derived from human monocytes and from mouse bone marrow as antigen-producing cells. They found that the nicotine induced expression by dendritic cells of co-stimulatory molecules such as . . . [Full Text of this Article]