Donate Help Contact The AHA Sign In Home
American Heart Association
Circulation
Search: search_blue_button Advanced Search
Circulation. 1976;54:795-800

This Article
Right arrow Full Text (PDF)
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrowRequest Permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Boucek, R. J.
Right arrow Articles by Boerth, R. C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Boucek, R. J., Jr
Right arrow Articles by Boerth, R. C.

Circulation, Vol 54, 795-800, Copyright © 1976 by American Heart Association


ARTICLES

Spontaneous resolution of massive congenital tricuspid insufficiency

RJ Boucek Jr, TP Graham Jr, JP Morgan, GF Atwood and RC Boerth

Four cases of congenital tricuspid insufficiency (CTI) are presented in whom spontaneous improvement occured. There was no evidence of Ebstein's disease in three of the four. All presented with similar clinical and laboratory findings including massive cardiomegaly, mild to moderate congestive heart failure, and moderate to severe desaturation. Catheterization data demonstrated severe TI and right-to- left atrial shunting. During the period of follow-up, up to 3.5 years, all symptoms have spontaneously disappeared. Clinical and angiocardiographic evidence for minimal residual TI have remained. Right atrial and right ventricular volume measurements were markedly abnormal initially and returned to normal or near normal. These measurements may be a useful method for quantitating the severity of tricuspid insufficiency. The course of the three infants with massive CTI (without Ebstein's disease) differs from previous reports and may be important both in formulating future management plans and in defining the etiology of the disorder.


This article has been cited by other articles:


Home page
HeartHome page
D Boshoff, L Mertens, and M Gewillig
Severe tricuspid regurgitation 14 years after diagnosis of ""transient neonatal tricuspid regurgitation""
Heart, July 1, 2001; 86(1): 88 - 90.
[Abstract] [Full Text] [PDF]


Home page
Arch Pediatr Adolesc MedHome page
A. L. Alkalay, D. A. Ferry, S. H. Pepkowitz, P. J. Chou, G. K. Oakes, and J. J. Pomerance
Critical Tricuspid Insufficiency due to Papillary Muscle Rupture: A Result of Prenatal Hypoxic Insult
Arch Pediatr Adolesc Med, July 1, 1988; 142(7): 753 - 755.
[Abstract] [PDF]