Circulation, Vol 54, 783-789, Copyright © 1976 by American Heart Association
BN Goldreyer, JA Kastor and KL Kershbaum
The circulatory effects of supraventricular tachycardia (SVT) were studied
in eight patients who reported disabling symptoms during paroxysms of the
arrhythmia. Supraventricular tachycardia was induced in each patient by
rapid atrial pacing or with atrial premature stimuli. Hemodynamic
parameters in sinus rhythm and following the initiation of SVT were
recorded and compared. The following mean values were observed in sinus
rhythm (SR) and SVT. Heart rate (beats/min): SR 79, SVT 183; P-R interval
(msec): during SR, 154; during SVT, 256; ratio of mean P-R intervals to
mean R-R cycl lengths: SR 20%, SVT 76%; brachial artery pressures (mmHg):
SR 141, SVT 99; cardiac index (L/min/m2): SR 3.6, SVT 2.2; pulmonary artery
pressures (mmHg): SR 18/7, SVT 26/15; peak right atrial pressures (mm Hg):
SR 4, SVT 17. Large waves appeared in the right atrium during SVT due to
atrial contraction against closed tricuspid valves. Pulsus alternans were
observed in each case during SVT. Despite the presence of chest pain during
SVT, the coronary arteries were normally patent in four patients who
underwent coronary arteriography.
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The hemodynamic effects of induced supraventricular tachycardia in man
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