Circulation, Vol 54, 237-244, Copyright © 1976 by American Heart Association
HJ Wellens, DR Duren and KI Lie
Fifty patients with sustained ventricular tachycardia were studied by
endocavitary recordings and programmed electrical stimulation. Of 29
patients in whom tachycardia could be initiated, 18 had chronic coronary
artery disease and eight had no detectable heart disease. Of 35 patients in
whom the tachycardia could be terminated by premature ventricular stimuli,
21 had chronic coronary artery disease and eight had no detectable heart
disease. Initiation of tachycardia was facilitated in 18 of 21 patients by
pacing the ventricle at the slowest possible pacing rate. An inverse
relation was found between the prematurity of the tachycardia-initiating
premature beat and the interval between the premature beat and the first
beat of tachycardia, a finding suggestive of a re-entry mechanism. The role
of the specific conduction system in initiation and during tachycardia
remains unknown. Stimulation site was found to affect initiation and
termination of tachycardia and width of tachycardia zone.
ARTICLES
Observations on mechanisms of ventricular tachycardia in man
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