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Genetically altered mice
Citations 311-320 of 480 total displayed.
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Past content
(since Jan 1999):
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- Articles
Robust Adenoviral and Adeno-Associated Viral Gene Transfer to the In Vivo Murine Heart. Application to Study of Phospholamban Physiology
- Hunter C. Champion, Dimitrios Georgakopoulos, Saptarsi Haldar, Lili Wang, Yibin Wang, and David A. Kass
Circulation published November 24, 2003, doi:10.1161/01.CIR.0000096487.88897.9B
[Abstract]
- Basic Science Reports
Aldosterone and Not Plasminogen Activator Inhibitor-1 Is a Critical Mediator of Early Angiotensin II/NG-Nitro-l-Arginine Methyl Ester-Induced Myocardial Injury
- Eveline M. Oestreicher, Diego Martinez-Vasquez, James R. Stone, Larus Jonasson, Weranuj Roubsanthisuk, Koji Mukasa, and Gail K. Adler
Circulation 2003;108:2517-2523; published online before print as doi:10.1161/01.CIR.0000097000.51723.6F
[Abstract]
[Full text]
- Articles
Common Genomic Response in Different Mouse Models of -Adrenergic-Induced Cardiomyopathy
- Vinciane Gaussin, James E. Tomlinson, Christophe Depre, Stefan Engelhardt, Christopher L. Antos, Gen Takagi, Lutz Hein, James N. Topper, Stephen B. Liggett, Eric N. Olson, Martin J. Lohse, Stephen F. Vatner, and Dorothy E. Vatner
Circulation published November 17, 2003, doi:10.1161/01.CIR.0000101922.18151.7B
[Abstract]
- Basic Science Reports
Opposing Effects Mediated by the Chemokine Receptor CXCR2 on Myocardial Ischemia-Reperfusion Injury: Recruitment of Potentially Damaging Neutrophils and Direct Myocardial Protection
- Sima T. Tarzami, Wenfeng Miao, Kartik Mani, Lillie Lopez, Stephen M. Factor, Joan W. Berman, and Richard N. Kitsis
Circulation 2003;108:2387-2392; published online before print as doi:10.1161/01.CIR.0000093192.72099.9A
[Abstract]
[Full text]
- Basic Science Reports
Inactivation of Macrophage Scavenger Receptor Class B Type I Promotes Atherosclerotic Lesion Development in Apolipoprotein EDeficient Mice
- Wenwu Zhang, Patricia G. Yancey, Yan Ru Su, Vladimir R. Babaev, Yuomin Zhang, Sergio Fazio, and MacRae F. Linton
Circulation 2003;108:2258-2263; published online before print as doi:10.1161/01.CIR.0000093189.97429.9D
[Abstract]
[Full text]
- Basic Science Reports
Reduction of Infarct Size and Prevention of Cardiac Rupture in Transgenic Mice Overexpressing FrzA
- Laurent Barandon, Thierry Couffinhal, Jérome Ezan, Pascale Dufourcq, Pierre Costet, Philippe Alzieu, Lionel Leroux, Catherine Moreau, Danièle Dare, and Cécile Duplàa
Circulation 2003;108:2282-2289; published online before print as doi:10.1161/01.CIR.0000093186.22847.4C
[Abstract]
[Full text]
- Articles
Aldosterone and Not Plasminogen Activator Inhibitor-1 Is a Critical Mediator of Early Angiotensin II/NG-Nitro-L-Arginine Methyl Ester-Induced Myocardial Injury
- Eveline M. Oestreicher, Diego Martinez-Vasquez, James R. Stone, Larus Jonasson, Weranuj Roubsanthisuk, Koji Mukasa, and Gail K. Adler
Circulation published October 27, 2003, doi:10.1161/01.CIR.0000097000.51723.6F
[Abstract]
- Articles
Inactivation of Macrophage Scavenger Receptor Class B Type I Promotes Atherosclerotic Lesion Development in Apolipoprotein E-Deficient Mice
- Wenwu Zhang, Patricia G. Yancey, Yan Ru Su, Vladimir R. Babaev, Yuomin Zhang, Sergio Fazio, and MacRae F. Linton
Circulation published October 27, 2003, doi:10.1161/01.CIR.0000093189.97429.9D
[Abstract]
- Articles
Reduction of Infarct Size and Prevention of Cardiac Rupture in Transgenic Mice Overexpressing FrzA
- Laurent Barandon, Thierry Couffinhal, Jérome Ezan, Pascale Dufourcq, Pierre Costet, Philippe Alzieu, Lionel Leroux, Catherine Moreau, Danièle Dare, and Cécile Duplàa
Circulation published October 27, 2003, doi:10.1161/01.CIR.0000093186.22847.4C
[Abstract]
- Articles
Opposing Effects Mediated by the Chemokine Receptor CXCR2 on Myocardial Ischemia-Reperfusion Injury. Recruitment of Potentially Damaging Neutrophils and Direct Myocardial Protection
- Sima T. Tarzami, Wenfeng Miao, Kartik Mani, Lillie Lopez, Stephen M. Factor, Joan W. Berman, and Richard N. Kitsis
Circulation published October 20, 2003, doi:10.1161/01.CIR.0000093192.72099.9A
[Abstract]
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